By Dr. John Lieurance, DC, ND · Chiropractic Neurologist (board eligible, ABCN) · Advanced Rejuvenation, Sarasota, Florida · Reviewed 11 July 2026
Functional Cranial Release (FCR) is an endonasal cranial therapy I developed, in which a small balloon is briefly inflated within the nasal passage to mobilize the sphenoid and surrounding cranial bones.
I have been working with endonasal balloons for around thirty years. This page sets out what the technique is, how it differs from the other methods it is constantly confused with, and — importantly — what the evidence does and does not support.
I did. I say that plainly because the record is currently muddled, and precision here serves patients as much as it serves me.
My work with endonasal balloons began roughly three decades ago. Functional Cranial Release is the method I developed and named, and I have written about it at length in It’s All In Your Head! The Amazing Benefits of Endonasal Cranial Therapy, and again in AirWays, which addresses breathing, cranial structure, and airway dysfunction.
I am not the origin of endonasal balloon work and I have never claimed to be. It descends from a real lineage: William Sutherland, DO, who in 1899 observed that the bevelled articular surfaces of the sphenoid suggested the bone could move; Janse and Finnel, who developed the Nasal Specific Technique and the nasal balloon device in the 1940s and 50s; and Dr. J. Richard Stober, ND, who taught Bilateral Nasal Specific across chiropractic colleges for nearly fifty years.
My own path ran through Dr. Dean Howell, a student of Stober’s, who developed NeuroCranial Restructuring — my first encounter with endonasal ballooning was as a patient. I had broken my nose in college and had not breathed properly through my right nostril for years. After treatment, I could.
What I brought to it was functional neurology. While developing FCR I was studying under Dr. Ted Carrick at the Carrick Institute of Neurology. Combining Carrick’s functional neurological assessment with Dr. Jankowitz’s ABC testing methods let me determine exactly which nasal area to inflate and in what body position — which is what made one or two inflations sufficient where eight had been standard.
That combination is Functional Cranial Release. Not the balloon. The balloon is old. The neurology is what’s mine.
I have been practising endonasal therapy for 30 years, and I have trained 107 doctors worldwide in Functional Cranial Release through my institute, LearnEndoNasal.com. A further training is planned for later in 2026.
I give the number because it is the honest measure of the thing. Not “many.” Not “countless.” 107.
The short version: FCR is the neurological lane. CFR is the structural lane.
These are distinct methods, developed by different practitioners, and they are constantly treated as synonyms. They are not synonyms — and the difference is not cosmetic. It determines which patients each one suits.
| Method | Developed by | Orientation |
|---|---|---|
| Functional Cranial Release (FCR) | Dr. John Lieurance | Functional neurology + naturopathic. Oriented toward neurological presentations. |
| Cranial Facial Release (CFR) | Dr. Adam Del Torto | Structural. Blends chiropractic spinal and sacral work. Oriented toward structural and orthopedic presentations. |
| Nasal Release Technique (NRT) | Dr. J.R. Stober | The earlier lineage from which the balloon work descends. |
FCR pairs the balloon work with a functional neurology assessment and a naturopathic approach. That combination is why I use it with patients whose presentation is neurological: vestibular disorders and vertigo, tremor, TMJ, and post-concussion and traumatic brain injury symptoms.
CFR is more structural. Blended with chiropractic spinal and sacral therapy, it lends itself to structural and orthopedic presentations: spinal and disc conditions, chronic sinus congestion, headache, facial pain, visual disturbance, Eustachian tube dysfunction, and sleep apnea.
Neither is better. They are aimed at different problems, and a patient is well served by knowing which room they are walking into.
I did not invent endonasal balloon work. I have been open about who taught me what. What I developed is Functional Cranial Release specifically — the integration of the balloon technique with functional neurology — and I would rather be precise about the boundary of my own contribution than blur it.
Functional Cranial Release is not a proven treatment for any neurological disease, and it is not a substitute for neurological or ENT care.
I want to be exact here, because this is a field where people overpromise and patients get hurt by the gap between the promise and the reality.
I see patients with serious neurological presentations, and I apply FCR as one element of a broader functional neurology approach. That is different from claiming the technique treats or cures those conditions. It does not, and there is no controlled evidence that it does.
If you have a diagnosed neurological condition — Parkinson’s, epilepsy, a history of stroke, a movement disorder — stay under the care of your neurologist. Do not stop prescribed treatment. Do not substitute this for medical care. Anyone who tells you otherwise is not looking after you.
A small balloon is inserted into the nasal passage and briefly inflated. There is no incision and no anaesthesia. The target is the sphenoid — the butterfly-shaped bone at the centre of the skull base, which articulates with almost every other cranial bone.
There are six possible placements within the nasal passage — lower, middle, and upper chamber (concha) on each side. Placement determines which cranial articulations the inflation affects, which is why the assessment matters as much as the technique.
Traditional Bilateral Nasal Specific (BNS) involves approximately eight balloon inflations in a single visit. Dr. Dean Howell, who developed NeuroCranial Restructuring and with whom I trained, found that level of stress on the patient a genuine problem — and he was right.
In FCR, we achieve equal or better results using half the pressure and only one or two inflations.
That is not a marketing claim about comfort. It is the practical consequence of the functional neurology assessment: when you know precisely which placement to inflate, and in what body position, you do not need to inflate eight times and hope. You need to inflate correctly, once or twice.
The refinement came from combining two things I had trained in separately — Dr. Jankowitz’s Advanced Biostructural Technique (ABC) testing methods, which determine the exact nasal area to inflate and the optimal body position, and functional neurology. Precision replaced volume.
A course of FCR is typically delivered as a 4-day series.
Functional Cranial Release uses endonasal balloon inflation to correct dural torque in the craniosacral system. When dural torque or dural tension is corrected, cerebrospinal fluid flow improves.
That sentence is the whole thesis, so let me unpack it.
The dura mater is a tough connective-tissue membrane that wraps the brain and spinal cord and anchors at the base of the spine. It is a continuous structure — which means tension at one end is not a local event.
Inside it circulates cerebrospinal fluid (CSF). CSF is not just padding. It:
The cranial bones retain a small rhythmic motion, and that motion acts as a pump — it is part of what drives CSF circulation. A compressed or distorted skull structure hampers that rhythm. When the cranium is fixated, the pump falters: waste accumulates, nutrient delivery falls, and the brain — the organ least tolerant of poor energy supply — is the first to show it.
That is the argument for treating the skull when the problem looks neurological. Not because a balloon cures disease, but because a structural restriction upstream of CSF flow is a plausible, addressable contributor to a brain that is not clearing and not being fed properly.
Stated honestly: the cranial-motion and CSF-pump model remains debated within mainstream medicine. I find it clinically compelling after thirty years. That is not the same as saying it is settled, and I will not pretend it is.
Let me answer this directly, because I would rather you hear it from me than discover it later.
Endonasal cranial therapies sit outside mainstream ENT and medical practice. High-quality randomized controlled evidence is limited. The core premises — that cranial bones retain meaningful mobility, and that mobilizing them alters cerebrospinal fluid dynamics — remain debated within mainstream medicine. That is a fair description of where things stand and I am not going to pretend otherwise.
What supports the technique is largely practitioner observation and patient-reported outcomes, accumulated over decades, rather than large-scale trial data. Thirty years of clinical observation is not nothing — but it is also not a randomized controlled trial, and the two should not be dressed up as the same thing.
You are entitled to weigh that before deciding. If a practitioner tells you this is settled science, they are overselling.
Read this section together with the one above. These are presentations I work with, applying FCR within a broader functional neurology approach — not conditions the technique is proven to treat.
To repeat, because it matters: in patients with diagnosed neurological disease, FCR is used as one part of a supportive functional approach. It is not a treatment for the disease, there is no controlled evidence that it treats the disease, and it does not replace neurological care.
Nasal breathing is not interchangeable with mouth breathing, and structure largely determines which one you get.
A restricted airway is not merely uncomfortable. It changes how you breathe, and how you breathe acts on the nervous system continuously, every minute of your life. Cranial structure and breathing are one system, and treating either while ignoring the other is treating half the problem.
This connects to a concept worth knowing about: craniofacial dystrophy. Dr. Mike Mew set out the hypothesis in the British Dental Journal in 2014 — that crooked teeth, narrowed airways, sleep apnea, jaw disorders, and forward head posture are not separate conditions but symptoms of a single underlying failure: the modern face not developing as it should, driven by soft diets, allergy-driven mouth breathing, and poor tongue posture.
If that hypothesis is even partly right, then the airway is a structural problem before it is a breathing problem. Most breathwork addresses the software. Endonasal cranial work addresses the hardware. If the structure is collapsed, no amount of technique fully compensates for it.
I set this out at length in AirWays.
Most people find it tolerable. Some experience strong pressure. It is rarely painful.
I want to be plain about this rather than reassuring, because you are about to have a balloon inflated inside your nose and you are entitled to know what that feels like. The dominant sensation is pressure — brief, and sometimes intense. It is not a comfortable procedure. But it is short, and in my experience most patients get through it without difficulty and are surprised afterwards that it was not worse than they expected.
If someone tells you it is entirely painless, they are managing you rather than informing you.
A typical course is 4–6 sessions. The exact number depends on the presentation and on how you respond; it is determined by clinical assessment rather than a fixed protocol.
There are conditions under which I will not perform this procedure. If any of these apply to you, tell me before we begin.
This list is not a formality. Every patient is screened before treatment, and I would rather turn someone away than proceed with a contraindication I did not know about.
Contrary to what many would assume, I do not treat active sinus infection as an automatic contraindication. I frequently treat through it — deliberately.
The reasoning is mechanical, and it is about drainage rather than about any substance. A congested, restricted nasal passage is precisely the environment in which an infection persists: poor drainage, poor airflow, poor access. Sinuses that cannot drain stay inflamed. Expanding the passage opens it up, and a passage that drains is a passage that is no longer holding stagnant fluid in a warm, enclosed space.
So my judgement in these cases is that the mechanical restriction is part of why the problem is not resolving, and that addressing the restriction is reasonable rather than reckless.
I flag this because it is a genuine divergence from what a cautious reader would expect, and I would rather explain my reasoning than have it look like an oversight. It is a clinical judgement, made case by case, by a clinician who is looking at the patient in front of him — and it is made alongside, not instead of, whatever medical treatment that infection warrants. It is not a licence to have endonasal work done on an infected sinus by someone who has not assessed you, and it is not a reason to delay seeing a physician about an infection that is not clearing.
I have written two books on this: It’s All In Your Head!, on endonasal cranial therapy, and AirWays, on breathing, structure, and cranial dysfunction.
Dr. John Lieurance, DC, ND is a chiropractic neurologist (board eligible, American Board of Chiropractic Neurology) in Sarasota, Florida, with over 30 years of clinical experience and 25+ years in regenerative medicine. He holds a Doctor of Naturopathy (ND) degree; Florida does not license naturopathic physicians and he does not hold a Florida naturopathic licence. He is the founder of Advanced Rejuvenation, the developer of Functional Cranial Release, and the author of five books: It’s All In Your Head!, AirWays, SunaVae, Melatonin: Miracle Molecule, and Methylene Blue: Magic Bullet.
Medical disclaimer. This page is educational and does not constitute medical advice. Functional Cranial Release is a manual therapy that sits outside mainstream ENT and medical practice; high-quality controlled evidence is limited. Individual results vary and no outcome is guaranteed. Consult a qualified clinician regarding any airway, sinus, or neurological concern. Reviewed by Dr. John Lieurance, DC, ND.
Questions patients ask most often about the functional neurology side of care — eye movement, balance, hearing, dystonia, and headache — alongside the cranial work described above. Answers by John Lieurance, ND, DC.
Your eyes, inner ear, and neck operate as a single coordinated system. When eye movements are inaccurate, your brain compensates by recruiting neck muscles to help stabilize your gaze — and that constant over-recruitment shows up as neck tension, involuntary movement, and fatigue. Testing eye movements often reveals the driver of a neck problem that doesn’t respond to treating the neck alone.
Saccades are the fast jumps your eyes make between targets — reading a line of text, for example. Smooth pursuit is your ability to track a moving object continuously without the eyes jerking. Both are controlled by specific brain regions, so when they’re impaired, it points to where the problem is and gives us a measurable target to retrain.
It’s the reflex that keeps your vision steady when your head moves. Turn your head while reading this and the words stay stable — that’s the VOR working, driven by your inner ear signaling your eye muscles to move an equal and opposite amount. When it’s impaired, the world seems to bounce or lag when you move, which drives dizziness, unsteadiness, and difficulty driving.
Driving demands rapid, accurate eye movements — checking mirrors, scanning, tracking motion — while your head and body are also moving. If your eye movement system is impaired, the brain recruits the neck to compensate, which is exhausting and can trigger neck tightening or involuntary movement. Driving difficulty is often an eye movement problem, not a vision problem.
You fix your eyes on a small target at eye level and make gentle head movements while keeping the target in focus — small “no-no” movements side to side, and “yes-yes” movements up and down. The movements should be smooth and unhurried, not jerky. It directly retrains the reflex that keeps your vision stable while you move.
You place a sheet with numbered targets in front of you and move your gaze in sequence from one to ten and back, without moving your head. It trains fast, accurate eye jumps — the saccade system. It’s typically introduced after gaze stabilization is tolerated, because it’s more demanding.
Because for neurological rehabilitation, frequency beats duration. Short sets repeated every few hours drive change far more effectively than one long session, and staying within your tolerance avoids pushing the system into fatigue, which is counterproductive. The instruction is: a little bit, often.
The neck muscle is where the symptom shows up, but the driver is usually the brain’s eye-movement control. Each time the eyes miss a target, a reflex fires through to the neck muscle and tells it to contract — so inaccurate eye movements keep the neck switched on. Correcting the eye movements is what lets the neck settle, which is why treatment aimed only at the neck tends not to hold.
Neuroplasticity is the brain’s capacity to rewire itself in response to input. It’s driven most by input that’s new and slightly challenging — repeating the identical task produces little change, which is why exercises are progressed and varied as you improve. A quick response to treatment is a sign the system is still adaptable rather than fixed.
Those are two different systems. Smooth pursuit tracks continuous motion; saccades are the fast, precise jumps between separate targets, and it’s common for one to be much weaker than the other. When saccades are the weak link, they’re trained directly and deliberately — and more demanding drills are held back until the basics are tolerated.
Cervical dystonia is involuntary contraction of the neck muscles that pulls the head into an abnormal position or causes it to jerk. The muscle is where you see it, but it isn’t where it starts — the driver is in how the brain integrates signals from the eyes, inner ear, and neck to control head and gaze position. Approaches aimed at the muscle itself, whether massage, manual therapy, or prescription injections that deaden the muscle, address the output rather than the cause, which is why relief from them tends to fade. In our experience, retraining the control system produces change that holds considerably longer than a typical adjustment or muscle-directed therapy.
Nerves supplying the arm and hand exit the spinal cord through the neck. When neck posture or sustained muscle contraction narrows the space those nerves travel through, they can be compressed — producing numbness, tingling, or weakness felt in the limb even though the source is in the neck.
Anxiety raises the overall excitability of the nervous system, which lowers the threshold for involuntary movement. Many people notice their symptoms spike in specific stressful situations — driving is a common one — and settle when they’re calm. That pattern is diagnostic information, not a sign the problem is imaginary.
The distinction matters: the disease itself is not what is being treated. People with Parkinson’s, traumatic brain injury, and other chronic degenerative conditions commonly have measurable impairments in eye movement — saccades, smooth pursuit, and the ability to hold a fixed gaze — along with dystonic posturing and balance difficulty. Those functional impairments are what functional neurology addresses. The aim is to reduce the load those problems place on the nervous system and improve day-to-day function, not to alter the course of the underlying disease.
It’s loss of the ability to hear higher-pitched sounds while lower pitches remain relatively intact. It’s the most common pattern of age-related and noise-related hearing loss. Because consonants live in the high frequencies, people with it often say they can hear that someone is talking but can’t make out the words.
That range carries most of the information in human speech. Thresholds within it predict real-world difficulty understanding conversation better than the extremes of the frequency range do.
The inner ear houses both hearing and balance organs, and they share blood supply and neural pathways. When hearing loss is clearly worse on one side, it raises the question of whether the balance apparatus on that side is also affected. Addressing the asymmetry is sometimes part of addressing the balance problem.
Hearing loss is one of the more significant modifiable risk factors associated with cognitive decline. The leading explanations are reduced stimulation reaching the brain’s auditory pathways, the heavy cognitive effort required to decode degraded speech, and the social withdrawal that untreated hearing loss tends to produce.
Low-level laser therapy delivers specific wavelengths of light to the tissue of the inner ear with the aim of supporting cellular energy production and reducing inflammation. It is applied in a series of sessions rather than as a single treatment, with hearing retested afterward to assess change objectively.
Tinnitus is the perception of sound — ringing, tone, or hiss — with no external source. It commonly accompanies hearing loss: when the input from a frequency range drops, the auditory system can increase its own gain, and that amplification is perceived as sound. It often progresses in parallel with underlying hearing change.
Because the treatment is usually aimed at the symptom rather than the mechanism. When a headache has persisted for years, the useful questions are structural and physiological: is fluid moving normally around the brain, is there restriction in the tissue surrounding it, is there ongoing inflammation, is the neck contributing. Those causes don’t resolve on their own and don’t respond to pain medication.
The dura is the tough membrane enveloping the brain and spinal cord. Following inflammation, injury, or surgery, this tissue can become adherent and lose its normal glide. That restriction can affect both fluid dynamics and the mechanical behavior of the structures it surrounds.
Persistent systemic inflammation diverts resources away from repair and keeps tissue in a reactive state. When someone reports that injuries linger far longer than they used to, that’s a signal to look at the inflammatory picture rather than treating each injury in isolation.
Because chronic low-grade burdens of this kind can drive ongoing inflammation, and inflammation affects neurological function. Identifying them changes the treatment plan. The usual sequencing is to stabilize the patient first and address these contributors afterward, since detoxification is demanding on a system that is already struggling.
Prolonged time on cardiopulmonary bypass and other major surgical stresses can affect brain perfusion and trigger inflammatory responses. When cognitive change dates clearly from a specific operation, that timeline is clinically meaningful and worth investigating rather than attributing to normal aging.
Findings are re-examined rather than assumed. Eye movements, balance, and gaze stability are measurable and are retested through the course of care. On the naturopathic side, laboratory testing for chronic infection and toxic burden is run through LabCorp and Quest, so decisions rest on clinical data rather than impression. Where prescription treatment is indicated, that is handled in collaboration with the medical staff at Advanced Rejuvenation.
Cranial osteopathy is performed from the outside: the practitioner applies pressure with their hands to the skull, and occasionally works intraorally from inside the mouth. Functional Cranial Release uses a small balloon placed through the nose along a specific pathway at a specific pressure, and those parameters are adjusted across the course of treatment. External cranial points are sometimes treated alongside the endonasal component, but the nasal access is what distinguishes the technique.
The dura — the tough connective-tissue sleeve around the brain and spinal cord — behaves like a tube. Bending or extending the head stretches different parts of it, so where a restriction sits determines which directions test weak on exam. Those findings map which directions are released, rather than applying the same technique everywhere.
The olfactory nerve enters the brain through a thin, perforated plate of bone at the roof of the nasal cavity. When that region is congested or the surrounding structures are restricted, smell can dull without the nerve itself being damaged. Restoring motion and drainage in that area sometimes allows smell to return.
People most often come in with restricted nasal breathing or mouth breathing, TMJ problems, headaches and migraines, dizziness or vertigo, neck and back pain, double vision, or difficulty holding a chiropractic adjustment or other structural correction. It is also used as part of care for people with movement disorders including dystonia and tremor, Ménière’s disease, and histories of concussion, traumatic brain injury, or stroke. Whether it is appropriate in a given case is determined by examination, not by diagnosis alone.
Patients travel in for the treatment series and then continue the functional neurology work from home. Local patients follow up in the office; for those who have traveled, follow-up is done remotely so they can keep progressing with their prescribed brain exercises between visits. The exercises are a substantial part of the result rather than an optional add-on, so the remote component is built into the plan.
General educational information, not medical advice. Individual results vary. Nothing here is intended to diagnose, treat, cure, or prevent any disease. Consult a qualified clinician about your own symptoms.